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e., Wdr77 is translocated within the nucleus into your cytoplasm to drive proliferation of prostate cancer cells. In this particular review, we Employed in vivo genetic research to more examine the purpose of Wdr77 in prostate tumorigenesis. We found that prostate-particular deletion of Wdr77 abolished prostate tumor initiation induced by loss of the tumor suppressor Pten. Mechanistically, Wdr77 ablation inhibited E2F3 activation and enhanced TGFβ signaling, leading to attenuated cellular proliferation induced by loss of Pten. These findings establish a vital part of Wdr77 for prostate tumor initiation.
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